FITC Anti-Mouse CD48 Antibody [HM48-1] (AGEL0695)

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SKU:
AGEL0695
Product Type:
Antibody
Antibody Type:
Monoclonal Antibody
Applications:
FC
Reactivity:
Mouse
Host Species:
Armenian Hamster
Isotype:
IgG2a,k
Clone:
HM48-1
Conjugation:
FITC
Frequently bought together:

Description

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FITC Anti-Mouse CD48 Antibody [HM48-1]

The Mouse CD48 Monoclonal Antibody (FITC Conjugated, HM48-1) provided by AssayGenie (Product Code: AGL0695) is an essential tool for researchers studying CD48, a transmembrane protein involved in immune responses and antigen presentation. This monoclonal antibody, designed for use in flow cytometry and immunofluorescence applications, specifically targets mouse CD48 and enables precise detection and analysis in various experimental settings.CD48, also known as BCM1 (B-lymphocyte activation marker), is a cell surface glycoprotein that plays a crucial role in mediating immune cell interactions and regulating immune responses. By binding to CD48, the HM48-1 monoclonal antibody allows researchers to investigate CD48 expression levels, distribution patterns, and functional roles in immune cells, making it a valuable resource for immunologists, cell biologists, and cancer researchers.

The FITC conjugation of this monoclonal antibody provides enhanced sensitivity and brightness, allowing for reliable and accurate detection of CD48 in flow cytometry experiments. Its high specificity and low background make it a versatile tool for investigating mouse CD48 expression in different cell types and biological samples, furthering our understanding of immune cell signaling and communication mechanisms.Overall, the Mouse CD48 Monoclonal Antibody (FITC Conjugated, HM48-1) from AssayGenie is a powerful research reagent that can facilitate cutting-edge studies in immunology, cancer biology, and beyond. Whether investigating immune cell interactions, studying antigen presentation pathways, or exploring immunomodulatory mechanisms, this antibody is sure to provide valuable insights and support diverse research endeavors.

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